including Phoenix metropolitan area, Scottsdale, Mesa, Chandler, Peoria, Tempe, Goodyear, Anthem, Cave Creek, Carefree, Apache Junction, Tucson and Sedona.

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IBR2 is a potent and specific RAD51 inhibitor and inhibits RAD51-mediated DNA double-strand break repair. IBR2 disrupts RAD51 multimerization, accelerates proteasome-mediated RAD51 protein degradation, inhibits cancer cell growth and induces apoptosis.

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[5] M C Aragoni, et al. Chemistry. Mechanistic aspects of the reaction between Br2 and chalcogenone donors (LE; E=S, Se): competitive formation of 10-E-3, T-shaped 1:1 molecular adducts, charge-transfer adducts, and [ (LE)2]2+ dications

[3] Jiewen Zhu, et al. EMBO Mol Med. A novel small molecule RAD51 inactivator overcomes imatinib-resistance in chronic myeloid leukaemia

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[2] Peter J Ferguson, et al. J Pharmacol Exp Ther. Synergistic Antiproliferative Activity of the RAD51 Inhibitor IBR2 with Inhibitors of Receptor Tyrosine Kinases and Microtubule Protein

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DIDS is a potent RAD51 inhibitor. DIDS inhibits the RAD51-mediated homologous pairing and strand-exchange reactions. DIDS inhibits anion exchange and binding to the red blood cell membrane.

CAM833 is a potent orthosteric inhibitor of the interaction between BRCA2 and RAD51 with a Kd of 366 nM against the ChimRAD51 protein. CAM833 also inhibits RAD51 oligomerization.